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Why Are Transgender People Immune To Optical Illusions?[Epistemic status: So, so speculative. Don’t take any of this seriously until it’s replicated and endorsed by other people.]I. If you’ve ever wanted to see a glitch in the Matrix, watch this spinning mask: Source: http: //hearingthevoice. Archives and past articles from the Philadelphia Inquirer, Philadelphia Daily News, and Philly.com. Did you see it? As the face started to turn away from you, your brain did…something, and then you were seeing a normal frontwards- facing mask again. It turns out your visual system has really strong views about whether faces should be inside- out or not, and it’s willing to execute a hard override on perception if it doesn’t like what it sees. But not always. Some people get glitchier glitches than others; a few seem almost immune. Studies find schizophrenics and autistic people to be consistently less glitchy than the rest of us. The correlation’s not perfect. But it’s definitely there.
![]() Something about these people’s different cognitive processing styles lets them see through the illusion. I wanted to replicate this result myself. So a few months ago, when I surveyed readers of my blog, I included some questions about perceptual illusions (including a static version of the hollow mask). I got five thousand responses, including a few from schizophrenic and autistic readers. Sure enough, the effect was there. Schizophrenic readers were about twice as likely to report a weak reaction to the mask illusion as non- schizophrenics (2. They were also more likely to have a weak reaction to a similar illusion, the Spinning Dancer (5. Readers with a family history of schizophrenia landed in between schizophrenics and healthy controls (1. Autistic readers were only slightly more likely to report a weak reaction to the mask illusion than neurotypicals (1. There was no different between autists and neurotypicals on the Spinning Dancer, not even a weak trend (5. Looking deeper, I found a few other anomalies on illusion perception. Most were small and inconsistent. But one stood out: transgender people had an altered response pattern on both illusions, stronger than the alteration for autism and almost as strong as the one for schizophrenia (mask: cis 1. These results are very tentative, and need replication. My mass survey isn’t a very sensitive instrument, and I place low confidence in any of this until other people can confirm. But for now, it sure looks like a signal. Something seems off about transgender people’s perception, something deep enough to alter the lowest- level components of visual processing. If it’s real, what could it be? II. A few days ago, trans blogger Zinnia Jones asked me if there might be any neurochemical reason trans people dissociate so much. Dissociation is a vague psychiatric symptom where you feel like you’re not real, or the world isn’t real, or you’re detached from the world, or something like that. It sounds weird, but if you explain it to someone who’s had it, they’ll say “Oh yeah, that thing!” It’s usually unpleasant, and tends to occur in PTSD, borderline personality, and extreme stress. And in transgender people. The only formal study I can find on this describes it as “greatly prevalent”, and suggests that up to 3. ![]() ![]() This matches trans people’s self- reports (1, 2, 3, 4, 5). Anecdotally (according to Zinnia’s impression of the trans community) and formally (see Costa & Colizzi 2. Intuitively this makes sense. Trans people feel like they’re “trapped in the wrong body”, so of course they feel detached from their bodies / like their bodies aren’t real / like their bodies aren’t theirs. Hormone therapy helps solve the “wrong body” problem, so it also solves the dissociative symptoms. We aim to bridge psychosocial and biological levels of explanation. We can say that someone is stressed out because their boss overworks them, but also because they’re secreting high levels of cortisol. We can say that someone is depressed because they broke up with their boyfriend, but also because they have decreased synaptogenesis in their hippocampus. Causation gets tricky, and this is a philosophical minefield for sure, but overall these two levels should be complementary rather than competitive. So what’s the biological correlate to trans people having dissociation problems? Practically all searches for the biological basis of dissociation end up at the NMDA glutamate receptor, one of the many neurotransmitter systems in the brain. Even though its cousins dopamine and serotonin usually get top billing, glutamate is probably the brain’s most important neurotransmitter, and NMDA glutamate receptors in particular are involved in all sorts of interesting things. Drugs that block NMDA receptors cause dissociation. The most famous dissociative anaesthetic, ketamine, is an NMDA antagonist. So is DXM, a recreational drug that causes dissociation in abusers. Wikipedia’s list of dissociative drugs is basically just fifty- five NMDA antagonists in a row. The only other category they list are kappa opioid agonists, and kappa opioid agonism probably – you guessed it – antagonize NMDA. If we take this result seriously, every substance we know of that causes dissociation is an NMDA antagonist in some way. Does anything improve NMDA function – an effect we might expect to alleviate dissociation? Yes, and among a list of intimidating research chemicals called things like “aminocyclopropanecarboxylic acid” is one familiar name: estrogen. See eg El- Bakri et al, which finds that “estrogen modulates NMDA receptors function in the brain…enhancing NMDA function”. Mc. Ewen et al: “One of the long- term effects of estradiol [estrogen] is to induce NMDA receptor binding sites in the CA1 region of the hippocampus.” Bi et al: “1. B- estradiol [estrogen] enhances NMDA receptor phosphorylation and function.” I don’t fully understand this research, but it seems to point to estrogen promoting NMDA activity in some way. So transgender people dissociate a lot, a state usually associated with hypofunctioning NMDA receptors. And trans women get better when they take estrogen, a hormone that improves NMDA function. That’s interesting. But what does this have to do with those optical illusions? III. The Hollow Mask illusion and its cousins may depend on NMDA function. To oversimplify: the brain interprets the world through Bayesian calculations. In Corlett et al’s model, it communicates top- down priors (ie assumptions based on previous knowledge about the world) through NMDA receptors and bottom- up new evidence through AMPA receptors. They write: In a hierarchical cortical system in which representations become more abstract with increasing distance from the primary input, higher levels of the hierarchy specify top- down predictions through NMDA receptor signaling and any mismatches between expectancy and experience are conveyed upward through the hierarchy via rapid AMPA and GABA signaling. When you see a hollow mask, the brute facts of how the mask looks are your bottom- up sensory evidence. Your top- down prior is that every other face you’ve seen for your entire life has been normal, not inside- out. Given the strength of the prior, the prior wins, and your brain interprets the mask as a normal face. Unless your brain is bad at applying priors, ie its NMDA receptors aren’t working that well. Then it just sticks with the bottom- up sensory evidence showing that the mask is hollow. Schizophrenia and autism both probably involve decreased NMDA function in different ways. For schizophrenia, see eg Olney, NMDA receptor hypofunction model of schizophrenia, and Coyle, NMDA receptor and schizophrenia: a brief history. Ketamine seems to replicate the symptoms of schizophrenia pretty well and is commonly used as a model for the disorder. For autism, see eg Lee, NMDA receptor dysfunction in autism spectrum disorders and this study where screwing with NMDA receptors in mice seems to turn them autistic. From this we would predict that estrogen would help treat schizophrenia and autism. It does. Schizophrenia is more common and more severe in men than women, with researchers noting that “gonadal steroids may play a role in buffering females against the development of schizophrenia”. Women are known to sometimes get schizophrenia triggered by menopause when their estrogen levels decrease. Estrogen supplementation is an effective schizophrenia treatment, and there’s some interest in developing estrogen receptor modulators that can help schizophrenic men without making them grow breasts. 9/29/2017 0 Comments How To Install Dock DavitsHow To Install Dock Davits For Sale
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